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Acetaldehyde ACA , an ethanol metabolite, exerts both stimulatory and depressive effects on isolated myocardial tissue, but its impact on individual cardiac myocytes is unknown. The purpose of this study was to determine whether ACA-induced myocardial depression is due to an intrinsic alteration of the contractile properties of heart at the cellular level. Mechanical properties of adult rat ventricular myocytes were evaluated using a video edge-detection system. Myocytes were electrically stimulated to contract at 0.
Neither the durations nor maximal velocities of shortening and relengthening were affected by ACA. These results suggest that ACA-induced myocardial depression in multicellular preparations is due to an intrinsic action on individual myocytes. Abstract Acetaldehyde ACA , an ethanol metabolite, exerts both stimulatory and depressive effects on isolated myocardial tissue, but its impact on individual cardiac myocytes is unknown.
Publication types Research Support, Non-U. Gov't Research Support, U. Gov't, P. Substances 3-Pyridinecarboxylic acid, 1,4-dihydro-2,6-dimethylnitro 2- trifluoromethyl phenyl -, Methyl ester Acetaldehyde Calcium.